Residual Force Enhancement Following Eccentric Contractions: A New Mechanism Involving Titin

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Residual Force Enhancement Following Eccentric Contractions: A New Mechanism Involving Titin.

Eccentric muscle properties are not well characterized by the current paradigm of the molecular mechanism of contraction: the cross-bridge theory. Findings of force contributions by passive structural elements a decade ago paved the way for a new theory. Here, we present experimental evidence and theoretical support for the idea that the structural protein titin contributes to active force prod...

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HIGHLIGHTED TOPIC Eccentric Exercise Mechanisms of enhanced force production in lengthening (eccentric) muscle contractions

Herzog W. Mechanisms of enhanced force production in lengthening (eccentric) muscle contractions. J Appl Physiol 116: 1407–1417, 2014. First published February 21, 2013; doi:10.1152/japplphysiol.00069.2013.—In contrast to isometric and shortening contractions, many observations made on actively lengthening muscles cannot be readily explained with the sliding filament and cross-bridge theory. Sp...

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Increased Residual Force Enhancement in Older Adults Is Associated with a Maintenance of Eccentric Strength

Despite an age-related loss of voluntary isometric and concentric strength, muscle strength is well maintained during lengthening muscle actions (i.e., eccentric strength) in old age. Additionally, in younger adults during lengthening of an activated skeletal muscle, the force level observed following the stretch is greater than the isometric force at the same muscle length. This feature is ter...

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Muscle residual force enhancement has been observed in different muscle preparations for more than half a century. Nonetheless, its mechanism remains unclear; to date, there are three generally accepted hypotheses: 1) sarcomere length non-uniformity, 2) engagement of passive elements, and 3) an increased number of cross-bridges. The first hypothesis uses sarcomere non-homogeneity and instabilit...

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ژورنال

عنوان ژورنال: Physiology

سال: 2016

ISSN: 1548-9213,1548-9221

DOI: 10.1152/physiol.00049.2014